General Details

Product Name: Acer truncatum seed oil
Plant Latin name: Acer truncatum Bunge
Production Method: Cold press
Appearance: Golden yellow oil

Health Benefits

Acer truncatum seed oil is cold-pressed from the plant seeds of Acer truncatum Bung, it is a rich source of nervonic acid, also known as selacholeic acid, and cis-15-tetracosenoic acid.

Nervonic acid is recognized by scientists worldwide as the only double-acting miracle substance that can repair and unblock the nerve fibers of damaged brain nerve pathways and promote the regeneration of nerve cells. Its lack will cause brain diseases such as sequela of apoplexy, Alzheimer’s disease, cerebral palsy, brain atrophy, memory loss, insomnia, and forgetfulness. The human body can’t produce nervonic acid and can only be supplemented by food intake.

Nervonic acid is an essential nutrient for developing brain function and visual function in brain organs. Infants and young children have a rapid nervous system development, and the brain weight is about 370 g at birth, 1000 g at two years old, and close to adult brain weight (about 1500 g) at seven years old.

The increase in brain weight is mainly due to the rise in the volume of nerve cells, the increase and lengthening of dendrites, and the formation and development of nerve myelin sheaths. Infants and young children who do not get enough nerve acid from food or breast milk are prone to growth retardation, visual impairment, and neurodevelopmental abnormalities, leading to mentally disabled children.

Young and middle-aged people are the period with the heaviest brain load. The function of the brain depends on the ability of brain cells to process information and transmit information. Adults’ lack of nervonic acid will cause memory loss, forgetfulness, and insomnia.

Nervonic acid is a double key factor of the brain cell membrane, and nerve fiber can enhance information between brain cells. Appropriate supplementation of nervonic acid can make you get rid of brain fatigue, memory decline, insomnia, forgetfulness, and other symptoms, improve thinking agility, response-ability, and work efficiency. It can also maintain vital energy and physical strength and prevent and effectively postpone brain function degeneration.

The common feature of encephalopathy, such as the sequelae of stroke in Alzheimer’s Parkinson’s disease, is that the patient’s myelin is destroyed, resulting in a severe decline in nervonic acid. The fundamental way to treat such encephalopathy is to adhere to a two-pronged approach and synchronize the treatment of brain nerve cells and nerve fibers.

Nervonic acid can induce nerve fibers to grow, repair, and dredge the blocked, distorted, condensed nerve fibers. It helps the brain pathway unblocked, restore some or all of the functions of Alzheimer’s Parkinson’s patients in terms of language, memory, sensory limbs, etc., and improve the main symptoms such as adverse speech after-stroke symptoms, hemiplegia, facial paralysis, and giddiness.

Clinical trials by the Shanghai Municipal Health Inspection Agency proved that, after taking nervonic acid for three months, the average efficiency of brain diseases was 96.6%, and the effective rate was 92.8%. The diseases include Parkinson’s cerebral palsy, cerebral atrophy, brain traumatic memory loss, and other brain diseases.

Adrenoleukodystrophy is an inherited lipid metabolic disease associated with insufficient nervonic acid and impaired myelin formation. Symptoms include physical weakness, inability to coordinate, vision and hearing loss, movement disorders, or low adrenal cortex function. Supplementation with nervonic acid significantly affects treating patients with adrenoleukodystrophy and can effectively help patients improve symptoms.

Multiple sclerosis is mainly the symptom of sphingomyelin damage to nerve fibers, the resulting loss of the block myelin. Its symptoms include limited mobility, unstable standing, impaired vision, pain, language obstruction, irritability, and insomnia.

Nervonic acid helps sphingosine esters and sphingomyelin synthesized in the human body, which promotes the myelination of nerve fibers, regenerates the myelin sheath that has fallen off its appearance, improves the hardening condition, and promotes the recovery of damaged nerve fibers.

Cerebral hepatic and renal syndrome (Zellweger syndrome) is a disease caused by peroxidase loss caused by the metabolism of long-chain fatty acids, forming a nervous system developmental delay, epilepsy, retinal disease, liver disease, and premature death. The intake of nervonic acid at the early stage can alleviate the above symptoms, such as liver dysfunction and cholestasis.